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Up to the development of complications (aortic insufficiency, cardiosclerosis, heart failure), syphilitic aortitis is almost asymptomatic.

If rheumatic aortitis is suspected, C-reactive protein, blood immunoglobulins, antinuclear antibodies, circulating immunocomplexes, etc. are examined. Serological blood tests (RPR test) or cerebrospinal fluid are necessary to confirm syphilitic aortitis. In case of tuberculous aortitis, a sputum examination for VC by PCR, a complete x-ray examination (lung x-ray, tomography) is indicated. Diagnosis of bacterial aortitis requires blood cultures for sterility.

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To find out the causes of damage to the aorta, patients with suspected aortitis should be consulted by a venereologist, rheumatologist, phthisiatrician, cardiologist. To substantiate the diagnosis of aortitis, it is necessary to study clinical, laboratory and instrumental data.

The data obtained are refined using ultrasound, aortography, CT or MSCT of the aorta. Differential diagnosis of aortitis is carried out with atherosclerosis of the aorta, coronary artery disease. Treatment of aortitis is inextricably linked with active therapy of the underlying disease. In infectious aortitis, antibiotics are the first-line drugs; with allergic aortitis - glucocorticoids, NSAIDs, immunosuppressants; with syphilitic aortitis - preparations of bismuth, iodine, antibiotics of the penicillin series. The effectiveness of therapy is monitored by the dynamics of clinical and laboratory parameters.

The presence of an aortic aneurysm, especially signs of its dissection, is the basis for consulting a vascular surgeon and angiosurgical treatment - aneurysm resection followed by aortic replacement. With the development of aortic stenosis, balloon dilation, stenting, or bypass may be required.

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The severity of the prognosis for aortitis is determined by its form and etiology.

The most serious prognosis for acute and subacute bacterial aortitis.

The course of syphilitic and tuberculous aortitis is the more favorable, the earlier specific treatment is started. The development of other forms of chronic aortitis is more dependent on the underlying disease. In the absence of treatment, the disease is prone to progression and complicated course.

For the prevention of aortitis, timely treatment of primary diseases, prevention of STDs, and active detection of tuberculosis are of paramount importance.

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Aortitis is inflammation of the aorta. Acute A. develops in connection with acute infectious diseases or traumatic injuries, chronic as a consequence of acute A., as well as on the basis of chronic infections, mainly syphilis, as well as Great Soviet Encyclopedia.

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Aortitis: signs of the disease, methods of diagnosis and treatment.

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Pathology is manifested by signs of inflammation itself and cardiological symptoms. The clinic depends on the origin of the disease and the location of the lesion of the aorta. Most often, pain occurs in the chest or abdomen, as well as signs of a violation of the general condition.

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The clinical picture is nonspecific, therefore, additional diagnostic methods are involved. Treatment is conservative. Often you have to deal with the complications of this pathology.

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The aorta is the largest vessel of the human body, but not only in diameter, but also in length. After leaving the heart in the chest, it passes to the abdomen. Therefore, the symptoms of Isotretinoin are different, and other specialists, in addition to cardiologists and vascular surgeons, deal with such patients.

Pathology can affect both all layers of the aorta, and individual ones. Therefore, allocate: endoortitis - damage to the inner layer of Accutane ; mesaortitis - a pathological process in the middle layer; periaortitis - an inflammatory process in the outer layer, which can very quickly spread to neighboring organs and tissues; panaortitis - damage to all layers of the aortic wall at once.

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All causes of aortitis are conditionally divided into two large groups: infectious; autoimmune (formally, they can be assigned the status of aseptic - those in which inflammation develops in a sterile environment, without the participation of pathogenic microorganisms).

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Nonspecific is considered pathogenic microflora, which can provoke the development of a number of infectious pathologies. These are usually bacterial pathogens. Of these, those infectious agents that in most cases are detected in various infectious organs and tissues most often lead to the development of aortitis.

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Specific is the microflora, the presence of which depends on the development of just one infectious disease. In this case, aortitis is a complication of an infectious lesion caused by specific pathogens. Most often it is: mycobacterium tuberculosis (Koch's wand) - cause tuberculosis of various localization (lungs, skin, intestines, and so on); pale treponema - provokes the development of syphilis (an infectious venereal disease).

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Aseptic inflammation of the aorta occurs, as a rule, against the background of an autoimmune lesion, which means that the recognition mechanism has failed, and the body perceives its own structures as foreign, as a result of which it begins to fight them by all means. As a rule, aortitis occurs with autoimmune pathologies: connective tissue; vessels.

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In this case, the development of aortitis should be expected against the background of such diseases as: ankylosing spondylitis - inflammation in the intervertebral joints with their subsequent fusion; rheumatoid arthritis - rheumatic damage (destruction) of the joints (mostly small); thromboangiitis obliterans is an autoimmune inflammation of small and medium-sized arterial and venous vessels.

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    Infectious aortitis occurs due to the introduction of pathogenic microflora into the wall of the aorta.

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    Accutane an introduction may be superficial, but inflammation develops as a reaction.an obvious process, therefore it is able to cover all layers of the aortic wall.

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    The infectious agent itself can enter its tissues:

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    According to the type of pathological process that develops in the tissues of the aortic wall, the following forms of the described disease are distinguished: purulent; necrotic; productive; granulomatous.

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    The purulent form occurs suddenly, acutely, the inflammatory process spreads both deep into the wall and over the surface.

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    The purulent process can quickly spread to neighboring tissues, causing a secondary purulent lesion.

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    A necrotic variety of aortitis appears on its own or against the background of acute purulent inflammation.

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    In this case, one or more areas of necrosis are formed. It is also characterized by an acute or subacute course.

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    Unlike the purulent form, it develops in isolation, does not directly spread to neighboring tissues and organs, but can provoke the development of reactive inflammation in them.

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    The productive inflammatory process is often chronic.

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    It develops slowly, may for some time be limited to one layer of the aorta, then cover others.

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